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These aforementioned statements somehow suggest that HSPs, presumably including all types and hsp60 and hsp70, may actually induce diseases, which may translate as a role in promoting cellular destruction and aging, instead of inhibiting these events. This conclusion, however, does not take into consideration the possibility that hsp70 and hsp60 may have other functions aside from possibly inducing aging and senescence. On the other hand, other findings suggest that HSPs are produced by aged cells only as a form of an “adaptive response” or as a way to “prevent protein aggregation and to assist in refolding, or degradation,” which are actually cellular reactions to the aging process (Soti & Csermely, “Molecular Chaperones” 227).